By C. C. Tijssen (auth.), M. Chatel, F. Darcel, J. Pecker (eds.)

The foreign assembly on mind Oncology in Rennes was once organised in honor of Jean Pecker, to pay tribute to his contribution to the advance of neurological sciences and to take inventory of the present country of data on mind tumors, a site within which the position of neurosurgery has been and should stay primordial. through the two-day convention, the foremost issues of mind oncology experiences, either primary and medical, have been tested: oncogenesis, tumoral markers and immunology, metabolic and analysis imaging, prognostic components and healing innovations. the big quantity and top of the range of participations led to a surely man made view of present advances in examine, of which this e-book offers the necessities. we've got tried either to maintain the richness of clinical exchanges which happened and to submit an outstanding many oral and poster communications. The publication respects the association of convention classes, and therefore displays the significance accorded to glial tumor experiences. this can look disproportionate given their frequency of incidence in share to overall numbers of intracranial tumor strategies, yet their gravity and quasi-total resistance to present healing equipment are considerable justification. additionally, this is often the area within which hopes of development are starting to seem, and it really is turning into attainable to envisage therapy in accordance with lately confirmed primary knowledge.

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Extra resources for Brain Oncology Biology, diagnosis and therapy: An international meeting on brain oncology, Rennes, France, September 4–5, 1986, held under the auspices of the Ministry of National Education, the University of Rennes and the Regional Hospital Rennes

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Double Minutes and Homogeneously Staining Regions: Gene Amplification in Mammalian Cells, Am Rev. Genet 16: 21-59 (1982). 14. 2. Cytogenet Cell Genet 40: 784 (1985). 15. ): Relationship Between Gene Amplification and Chromosomal Deviations in Malignant Human Gliomas. Submitted (1986). 16. ), HARSH (I. ): Numeric and Structural Alterations of Chromosome 7 in Human Brain Tumors: Correlation With Expression of Epidermal Growth Factor Receptor (EGFR). Proc. AACR 27: 37 (1986). 17. ): Polysomy of Chromosome 7 is Correlated With Overexpression of the erbB Oncogene in Human Glioblastoma Cell Lines.

G. Broggi, A. Franzini, S. Ferraresi, C. Giorgi, A. Allegranza 75-78 PROLIFERATIVE POTENTIAL OF MALIGNANT AND NON-MALIGNANT MENINGIOMAS. G. Cho, T. Hoshino, T. L. Davis 79-81 HISTOLOGICAL PATTERNS IN GERM CELL TUMORS. J. Verlooy, P. Cras, P. J. Martin DYSONTOGENETIC BRAIN TUMOURS; MORPHOLOGICAL VARIABILITY AND PROBLEMS OF CLASSIFICATION. F. Walter CONGENITAL INTRACRANIAL TUMORS. J. H. Goebel, S. Al-Hami, D. Voth, P. Gutjahr MENINGIOMAS: THE VALUE OF DIAGNOSTIC CYTOLOGY BY TISSUE IMPRINTS. U. Simi, R.

Certain glioblastomas and gliosarcomas which contain compact foci of small undifferentiated epithelial-like cells may demonstrate, in the same foci, immunopositivity for cytokeratin (19). In the same keratin-positive areas, clear-cut squamous differentiation may also be recognized in routine stains. Neuron-specific enolase, a cytoplasmic enzyme of the glycolytic cycle, therefore a non-structural marker, provides another instance in which the protein may be expressed in neoplastic cells and not in their normal equivalents.

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